Please use this identifier to cite or link to this item: https://hdl.handle.net/10316/114786
Title: Cardiovascular and Cerebrovascular Response to RedBull® Energy Drink Intake in Young Adults
Authors: Costa, Rita
Rocha, Clara 
Santos, Helder 
Keywords: Energy drink; carotid arteries; middle cerebral artery; peak systolic velocity; endodiastolic velocity
Issue Date: Jan-2023
Publisher: Kare Publishing
Project: UIDB/00308/2020 
Serial title, monograph or event: Anatolian Journal of Cardiology
Volume: 27
Issue: 1
Abstract: Background: Energy drinks contain caffeine, taurine, sucrose, vitamins, and other amino acids. The dosage of these varies depending on the drink chosen. Several studies on energy drinks have been carried out, but the results obtained are still inconsistent as well as the risk associated with consumption. This study analyzed the cardio- and cerebrovascular responses after consumption of an energy drink – RedBull® – under standardized pre- and post-ingestion conditions and its impact on the cardiovascular and cerebrovascular system. Methods: A sample of 30 healthy young adult females was recruited and subjected to 3 moments of evaluation: at baseline, 30 minutes after ingesting the energy drink, and 60 minutes after ingesting it according to a non-randomized pre–post intervention study design. Results: It is found that over time there are significant changes in peak systolic velocity (P = .006) and endodiastolic velocity (P < .001) of common carotid artery, peak systolic velocity (P = .007), and endodiastolic velocity (P < .001) of internal carotid artery, peak systolic velocity (P = .004), end endodiastolic velocity (P = .013) of the external carotid artery, endodiastolic velocity (P = .042) of the middle cerebral artery, cardiac output (P = .004), and heart rate (P < .001). Conclusions: After the consumption of Redbull®, there was a decrease in the velocities of the carotid arteries and the middle cerebral artery as well as a decrease in cardiac output accompanied by a decrease in heart rate and a slight, although not significant, increase in systolic and diastolic blood pressures. However, it is still unclear which pathophysiological mechanisms are responsible for these changes.
URI: https://hdl.handle.net/10316/114786
ISSN: 21492263
DOI: 10.14744/AnatolJCardiol.2022.2315
Rights: openAccess
Appears in Collections:I&D INESCC - Artigos em Revistas Internacionais
FCTUC Eng.Electrotécnica - Artigos em Revistas Internacionais

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