Please use this identifier to cite or link to this item:
https://hdl.handle.net/10316/95084
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Veloso, Caroline D. | - |
dc.contributor.author | Belew, Getachew D. | - |
dc.contributor.author | Ferreira, Luciana L. | - |
dc.contributor.author | Grilo, Luís F. | - |
dc.contributor.author | Jones, John Griffith | - |
dc.contributor.author | Portincasa, Piero | - |
dc.contributor.author | Sardão, Vilma A. | - |
dc.contributor.author | Oliveira, Paulo J. | - |
dc.date.accessioned | 2021-06-21T12:12:44Z | - |
dc.date.available | 2021-06-21T12:12:44Z | - |
dc.date.issued | 2019 | - |
dc.identifier.issn | 13816128 | pt |
dc.identifier.uri | https://hdl.handle.net/10316/95084 | - |
dc.description.abstract | Cardiovascular diseases (CVDs) are a leading risk factor for mortality worldwide and the number of CVDs victims is predicted to rise through 2030. While several external parameters (genetic, behavioral, environmental and physiological) contribute to cardiovascular morbidity and mortality; intrinsic metabolic and functional determinants such as insulin resistance, hyperglycemia, inflammation, high blood pressure and dyslipidemia are considered to be dominant factors. | pt |
dc.language.iso | eng | pt |
dc.relation | PTDC/DTP-FTO/2433/2014 | pt |
dc.relation | POCI-01-0145-FEDER-016659 | pt |
dc.relation | POCI-01-0145-FEDER-007440 | pt |
dc.relation | Marie Skłodowska-Curie Grant Agreement No. 722619 (FOIE GRAS) | pt |
dc.rights | embargoedAccess | pt |
dc.rights.uri | http://creativecommons.org/licenses/by/4.0/ | pt |
dc.subject | Cardiovascular diseases; Mitochondria; cardiovascular risk; insulin resistance; metabolism; mitochondrial dysfunction. | pt |
dc.subject.mesh | Animals | pt |
dc.subject.mesh | Cardiovascular Diseases | pt |
dc.subject.mesh | Dyslipidemias | pt |
dc.subject.mesh | Humans | pt |
dc.subject.mesh | Hyperglycemia | pt |
dc.subject.mesh | Hypertension | pt |
dc.subject.mesh | Inflammation | pt |
dc.subject.mesh | Insulin Resistance | pt |
dc.subject.mesh | Mitochondria | pt |
dc.subject.mesh | Oxidative Stress | pt |
dc.subject.mesh | Rats | pt |
dc.subject.mesh | Reactive Oxygen Species | pt |
dc.subject.mesh | Risk Factors | pt |
dc.title | A Mitochondrial Approach to Cardiovascular Risk and Disease | pt |
dc.type | article | - |
degois.publication.firstPage | 3175-3194 | pt |
degois.publication.lastPage | 3194 | pt |
degois.publication.issue | 29 | pt |
degois.publication.title | Current Pharmaceutical Design | pt |
dc.relation.publisherversion | https://www.eurekaselect.com/174592/article | pt |
dc.peerreviewed | yes | pt |
dc.identifier.doi | 10.2174/1389203720666190830163735 | pt |
degois.publication.volume | 25 | pt |
dc.date.embargo | 2019-06-30 | * |
uc.date.periodoEmbargo | 180 | pt |
item.openairecristype | http://purl.org/coar/resource_type/c_18cf | - |
item.grantfulltext | open | - |
item.openairetype | article | - |
item.languageiso639-1 | en | - |
item.fulltext | Com Texto completo | - |
item.cerifentitytype | Publications | - |
crisitem.project.grantno | info:eu-repo/grantAgreement/EC/H2020/722619/EU/Bioenergetic Remodeling in the Pathophysiology and Treatment of Non-Alcoholic Fatty Liver Disease | - |
crisitem.author.researchunit | CNC - Center for Neuroscience and Cell Biology | - |
crisitem.author.researchunit | CNC - Center for Neuroscience and Cell Biology | - |
crisitem.author.researchunit | CNC - Center for Neuroscience and Cell Biology | - |
crisitem.author.orcid | 0000-0002-3745-3885 | - |
crisitem.author.orcid | 0000-0001-7014-4614 | - |
crisitem.author.orcid | 0000-0002-5201-9948 | - |
Appears in Collections: | I&D CNC - Artigos em Revistas Internacionais |
Files in This Item:
File | Description | Size | Format | |
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2019.07.22_Final paper Veloso et al.pdf | 962.85 kB | Adobe PDF | View/Open |
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